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Moreover, neuropathological examination of MS tissue indicates that degeneration, CNS atrophy, and myelin loss are most prominent in the progressive stage, when lymphocytic inflammation paradoxically wanes

This is not merely additive: the MOTS-c may amplify or extend the mitochondrial biogenesis signal beyond what training alone achieves, particularly in older adults whose endogenous MOTS-c response to exercise is blunted [2]

We hold both because regenerative medicine work demands that level of rigor

Research suggests they might work through sequential and complementary pathways: BPC-157 could provide initial cytoprotection and anti-inflammatory effects, creating a favorable environment for repair, while TB-500 might then promote cellular migration and structural reorganization